Slash Fracture Risk 49%

If you’re over 50, you should know THIS about your bones: If you’re not taking steps to make them stronger, they’re likely getting weaker by the day.

That’s because once you pass 50, your bones break down faster than they form. This imbalance can lead to a loss of bone density, the development of osteoporosis, and, ultimately, an increased risk of fractures.

There are numerous ways to build bone density—exercise and boosting your calcium are two of the best known.

But there’s ANOTHER critical vitamin you should know about that’s shown to SLASH fractures by 49 percent!

Popeye the Sailorman is synonymous with spinach… and strong muscles. But it turns out spinach can also power up another part of your body: your bones.

Researchers looked at over 1,400 women and found that those who ate 100 micrograms (mcg) of spinach per day were 31 percent less likely to suffer a fracture than those who ate about 60 mcg.

You’d need to eat about one to two servings of the greens daily to reach that goal.

Looking at hip fractures specifically, those who ate more spinach cut their risk of being hospitalized with a fracture by nearly half.

That’s a BIG deal since hip fractures, in particular, are associated with a higher risk of disability and death.

What’s so special about spinach? It’s loaded with the bone-building nutrient vitamin K1.

Spinach is an extremely versatile vegetable. You can add it to your smoothies, put it in your omelets, sauté it with butter and garlic, or use it as a base for a salad.

But if spinach isn’t your thing, don’t worry. You can also get K1 from Brussels sprouts, cabbage, and broccoli.

Who knew protecting your bones could be so easy?

P.S. If you want additional bone protection new research suggests the solution can be found in a most unlikely place. Click here for the surprisingly delicious scoop.

SOURCE:

“Dietary Vitamin K1 intake is associated with lower long-term fracture-related hospitalization risk: the Perth longitudinal study of ageing women.” Food & Function, 2022; 13 (20): 10642 DOI: 10.1039/D2FO02494B