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There’s not much good news when it comes to Parkinson’s disease.

It impacts more than 10 million people around the world.

There is NO CURE.

And current treatment only focuses on symptoms… not on actually stopping its progression.

The picture is bleak, but there is some good news…

Researchers have identified a substance that can neutralize a key enzyme involved in Parkinson’s… and it’s found in a common, inexpensive vitamin.

Although most cases of Parkinson’s disease are not hereditary, 15% of cases are passed to children from their parents through genetic mutations.

In those cases, the main culprit is a mutation in a gene that encodes what’s known as the dardarina enzyme (which is the Basque word for tremor). Technically, it’s called the LRRK2 enzyme.

Scientists have been on the search for a way to inhibit this enzyme.

And just recently, an international research team has finally found what they’ve been looking for in an unlikely place.

It’s in an active form of vitamin B12 called AdoCbl.

Studies in human cells and brain tissue show that AdoCbl inhibits the harmful activity of the dardarina enzyme, while also preventing the neurotoxicity caused by it.

That’s an exciting one-two punch!

This isn’t the first study to show a connection between vitamin B12 and Parkinson’s.

Other studies show that people with Parkinson’s tend to have lower levels of B12, and the disease also progresses more rapidly in those with lower levels than in those with higher levels.

Even better?

Supplementing with B12 can dramatically slow the progression of the disease, impacting factors like motor skills, behavior, and mood.

As always, this latest study will be used as a jumping off point to make drugs aimed at capitalizing on the active form of B12, AdoCbl.

That could be a long way off, and who knows what kind of side effects their synthetic version will come with.

In the meantime, I recommend anyone with Parkinson’s talking to their doctor about getting their B12 levels checked, and beginning supplementation if they’re low.


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